Browsing by Author "Pelegrín, Pablo"

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  • Tapiz I Reula, Alfonso José; Cochino, Alexis-Virgil; Martins, Andreia L.; Angosto-Bazarra, Diego; Ortiz de Landazuri, Iñaki; Mensa Vilaró, Anna; Cabral, Marta; Baroja-Mazo, Alberto; Baños, María C.; Lobato-Salinas, Zulema; Fabregat, Virginia; Plaza, Susana; Yagüe, Jordi L.; Casals López, Ferran; Oliva Miguel, Baldomero; Figueiredo, Antonio E.; Pelegrín, Pablo; Aróstegui Gorospe, Juan Ignacio (SpringerOpen, 2022)
    Pathogenic RIPK1 variants have been described as the cause of two different inborn errors of immunity. Biallelic loss-of-function variants cause the recessively inherited RIPK1 deficiency, while monoallelic variants impairing ...
  • Ionescu, Daniela; Peñín Franch, Alejandro; Mensa Vilaró, Anna; Castillo, Paola; Hurtado Navarro, Laura; Molina-López, Cristina; Romero-Chala, Silvia; Plaza, Susana; Fabregat, Virginia; Buján, Segundo; Marques, Joana; Casals López, Ferran; Yagüe, Jordi L.; Oliva Miguel, Baldomero; Fernández-Pereira, Luis Miguel; Pelegrín, Pablo; Aróstegui Gorospe, Juan Ignacio (Wiley, 2022)
    Objective: Autoinflammatory diseases are inherited disorders of innate immunity that usually start during childhood. However, several recent reports have described an increasing number of patients with autoinflammatory ...
  • Martín-Sánchez, Fátima; Martínez-García, Juan José; Muñoz-García, María; Martínez-Villanueva, Miriam; Noguera-Velasco, José A.; Andreu Martínez, David; Rivas, Luis; Pelegrín, Pablo (Springer Nature, 2017)
    The nucleotide-binding domain and leucine-rich repeat-containing receptor with a pyrin domain 3 (NLRP3) inflammasome is a sensor for different types of infections and alterations of homeostatic parameters, including ...
  • Tapia-Abellán, Ana; Angosto-Bazarra, Diego; Alarcón-Vila, Cristina; Baños, María C.; Hafner-Bratkovič, Iva; Oliva Miguel, Baldomero; Pelegrín, Pablo; Pelegrín, Pablo (American Association for the Advancement of Science (AAAS), 2021)
    The NLRP3 inflammasome is activated by a wide range of stimuli and drives diverse inflammatory diseases. The decrease of intracellular K+ concentration is a minimal upstream signal to most of the NLRP3 activation models. ...

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