Browsing by Author "Mato, Susana"

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  • CB1 knockout mice display impaired functionality of 5-HT1A and 5-HT2A/C receptors
    Mato, Susana; Aso Pérez, Ester; Castro, Elena; Martín, Miguel; Valverde Granados, Olga; Maldonado, Rafael, 1961-; Pazos, Ángel (Wiley-Blackwell, 2007)
    Interaction between brain endocannabinoid (EC) and serotonin (5-HT) systems was investigated by examining 5-HT-dependent behavioural and biochemical responses in CB1 receptor knockout mice. CB1 knockout animals exhibited ...
  • Possible therapeutic doses of cannabinoid type 1 receptor antagonist reverses key alterations in fragile X syndrome mouse model
    Gomis González, Maria, 1988-; Busquets Garcia, Arnau, 1985-; Matute, Carlos; Maldonado, Rafael, 1961-; Mato, Susana; Ozaita Mintegui, Andrés, 1969- (MDPI, 2016)
    Fragile X syndrome (FXS) is the most common monogenetic cause of intellectual disability. The cognitive deficits in the mouse model for this disorder, the Fragile X Mental Retardation 1 (Fmr1) knockout (KO) mouse, have ...
  • Synaptic plasticity and spatial working memory are impaired in the CD mouse model of Williams-Beuren syndrome
    Borralleras Fumaña, Cristina, 1988-; Mato, Susana; Amédée, Thierry; Matute, Carlos; Mulle, Christophe; Pérez Jurado, Luis Alberto; Campuzano Uceda, María Victoria (BioMed Central, 2016)
    Mice heterozygous for a complete deletion (CD) equivalent to the most common deletion found in individuals with Williams-Beuren syndrome (WBS) recapitulate relevant features of the neurocognitive phenotype, such as ...
  • Targeting the endocannabinoid system in the treatment of fragile X syndrome
    Busquets Garcia, Arnau, 1985-; Gomis González, Maria, 1988-; Guegan, Thomas, 1983-; Agustín Pavón, Carmen; Pastor, Antonio; Mato, Susana; Pérez Samartín, A.; Matute, Carlos; Torre Fornell, Rafael de la; Dierssen, Mara; Maldonado, Rafael, 1961-; Ozaita Mintegui, Andrés, 1969- (Nature Publishing Group, 2013)
    Fragile X syndrome (FXS), the most common monogenic cause of inherited intellectual disability and autism, is caused by the silencing of the FMR1 gene, leading to the loss of fragile X mental retardation protein (FMRP), a ...

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