Browsing by Author "Lleó, Alberto"

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  • Calabria, Marco; Hernández Pardo, Mireia; Cattaneo, Gabriele, 1984-; Serras Robert, Mariona; Juncadella, Montserrat; Reñé, Ramón; Sala, Isabel; Lleó, Alberto; Ortiz-Gil, Jordi; Ugas, Lidia; Ávila, Asunción; Gómez Ruiz, Isabel; Ávila, César; Costa, Albert, 1970- (Elsevier, 2020)
    Lifelong bilingualism may contribute to cognitive reserve (CR) in neurodegenerative diseases as shown by a delay of the age at symptom onset in bilinguals with Alzheimer’s disease (AD) and Mild Cognitive Impairment (MCI). ...
  • Lleó, Alberto; Chiva, Cristina; Sabidó Aguadé, Eduard, 1981-; Belbin, Olivia (Elsevier, 2019)
    A biomarker of synapse loss, an early event in Alzheimer's disease (AD) pathophysiology that precedes neuronal death and symptom onset, would be a much-needed prognostic biomarker. With direct access to the brain interstitial ...
  • Fortea, Juan; González-Ortiz, Sofía; Lleó, Alberto (Elsevier, 2020)
    Background: Alzheimer's disease and its complications are the leading cause of death in adults with Down syndrome. Studies have assessed Alzheimer's disease in individuals with Down syndrome, but the natural history of ...
  • Calabria, Marco; Cattaneo, Gabriele, 1984-; Marne, Paula; Hernández Pardo, Mireia; Juncadella, Montserrat; Gascón-Bayarri, Jordi; Sala, Isabel; Lleó, Alberto; Ortiz-Gil, Jordi; Ugas, Lidia; Blesa, Rafael; Reñé, Ramón; Costa, Albert, 1970- (Elsevier, 2017)
    In the context of bilingual research, little is known about the effects of neurodegenerative disorders (NDs) on the processing of two languages in a bilingual. In a recent cross-sectional study, we showed that Mild Cognitive ...
  • Spataro, Nino, 1984-; Calafell i Majó, Francesc; Cervera-Carles, Laura; Casals López, Ferran; Pagonabarraga, Javier; Pascual Sedano, Berta; Campolongo Perillo, Antònia; Kulisevsky, Jaime J.; Lleó, Alberto; Navarro i Cuartiellas, Arcadi, 1969-; Clarimón Echevarría, Jordi; Bosch Fusté, Elena (Oxford University Press, 2015)
    Parkinson's disease (PD) can be divided into familial (Mendelian) and sporadic forms. A number of causal genes have been discovered for the Mendelian form, which constitutes 10-20% of the total cases. Genome-wide association ...
  • Guix Ràfols, Francesc Xavier; Wahle, Tina; Vennekens, Kristel; Snellinx, An; Chávez Gutiérrez, Lucía; Ill-Raga, Gerard, 1982-; Ramos Fernández, Eva, 1984-; Guardia Laguarta, Cristina; Lleó, Alberto; Arimon, Muriel; Berezovska, Oksana; Muñoz López, Francisco José, 1964-; Dotti, Carlos G.; De Strooper, Bart (Wiley-VCH Verlag, 2012)
    Inherited familial Alzheimer's disease (AD) is characterized by small increases in the ratio of Aβ42 versus Aβ40 peptide which is thought to drive the amyloid plaque formation in the brain of these patients. Little is known ...
  • García-Sánchez, Carmen; Calabria, Marco; Grunden, Nicholas; Pons, Catalina; Arroyo-Díaz, Juan Antonio; Gómez-Anson, Beatriz; Lleó, Alberto; Alcolea, Daniel; Belvís, Roberto; Morollón, Noemí; Mur, Isabel; Pomar, Virginia; Domingo, Pere (Wiley, 2022)
    Background: While much of the scientific focus thus far has been on cognitive sequelae in patients with severe COVID-19, subjective cognitive complaints are being reported across the spectrum of disease severity, with ...
  • Lleó, Alberto; Medrano-Martorell, Santiago; Fortea, Juan (Nature Research, 2021)
    Plasma tau phosphorylated at threonine 181 (p-tau181) predicts Alzheimer's disease (AD) pathology with high accuracy in the general population. In this study, we investigated plasma p-tau181 as a biomarker of AD in individuals ...
  • Ramos Fernández, Eva, 1984-; Tajes Orduña, Marta; Palomer, Ernest; Ill-Raga, Gerard, 1982-; Bosch Morató, Mònica, 1986-; Guivernau Almazán, Biuse, 1988-; Roman Degano, Irene; Eraso Pichot, Abel; Alcolea, Daniel; Fortea, Juan; Núñez, Laura; Páez, Antonio; Alameda Quitllet, Francisco; Fernàndez Busquets, Xavier; Lleó, Alberto; Elosua Llanos, Roberto; Boada, Mercè; Valverde, M. A. (Miguel Ángel), 1963-; Muñoz López, Francisco José, 1964- (IOS Press, 2014)
    Glycation and nitrotyrosination are pathological posttranslational modifications that make proteins prone to losing their physiological properties. Since both modifications are increased in Alzheimer's disease (AD) due to ...
  • Lleó, Alberto; Suárez-Calvet, Marc (Wolters Kluwer (LWW), 2021)

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