Visualitza per autoria "Giralt, Albert"

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  • Targeting CAG repeat RNAs reduces Huntington's disease phenotype independently of huntingtin levels.
    Rué, Laura; Bañez Coronel, Mónica; Creus-Muncunill, Jordi; Giralt, Albert; Alcalá-Vida, Rafael; Mentxaka, Gartze; Kagerbauer, Birgit; Zomeño-Abellán, M. Teresa; Aranda, Zeus; Venturi, Veronica; Pérez-Navarro, Esther; Estivill, Xavier, 1955-; Martí, Eulàlia (American Society for Clinical Investigation, 2016)
    Huntington's disease (HD) is a polyglutamine disorder caused by a CAG expansion in the Huntingtin (HTT) gene exon 1. This expansion encodes a mutant protein whose abnormal function is traditionally associated with HD ...
  • TUNAR lncRNA encodes a microprotein that regulates neural differentiation and neurite formation by modulating calcium dynamics
    Senís, Elena; Esgleas, Miriam; Najas Sales, Sònia, 1985-; Jiménez-Sábado, Verónica; Bertani, Camilla; Giménez-Alejandre, Marta; Escriche, Alba; Ruiz Orera, Jorge, 1988-; Hergueta-Redondo, Marta; Jiménez, Mireia; Giralt, Albert; Nuciforo, Paolo G.; Albà Soler, Mar; Peinado, Héctor; Toro, Daniel; Hove-Madsen, Leif; Götz, Magdalena; Abad, María (Frontiers, 2021)
    Long noncoding RNAs (lncRNAs) are regulatory molecules which have been traditionally considered as "non-coding". Strikingly, recent evidence has demonstrated that many non-coding regions, including lncRNAs, do in fact ...

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